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MET

MET proto-oncogene, receptor tyrosine kinase
General Information
Name MET proto-oncogene, receptor tyrosine kinase
Alias
  • hepatocyte growth factor receptor
  • HGF receptor
  • HGF/SF receptor
  • SF receptor
  • proto-oncogene c-Met
  • scatter factor receptor
  • tyrosine-protein kinase Met
Gene_family Receptor tyrosine kinases|IPT domain containing
organism Homo sapiens
entrez_id 4233
location 7q31
transcript_count 6
exon_count 22
Location
7p22.3p21.3p15.2p14.1p12.2q11.1q11.23q21.12q22.1q31.1q31.33q34q36.2
by NCBI GRCh38.p14
Summary
    Entrez This gene encodes a member of the receptor tyrosine kinase family of proteins and the product of the proto-oncogene MET. The encoded preproprotein is proteolytically processed to generate alpha and beta subunits that are linked via disulfide bonds to form the mature receptor. Further processing of the beta subunit results in the formation of the M10 peptide, which has been shown to reduce lung fibrosis. Binding of its ligand, hepatocyte growth factor, induces dimerization and activation of the receptor, which plays a role in cellular survival, embryogenesis, and cellular migration and invasion. Mutations in this gene are associated with papillary renal cell carcinoma, hepatocellular carcinoma, and various head and neck cancers. Amplification and overexpression of this gene are also associated with multiple human cancers. [provided by RefSeq, May 2016]
    Stringdb Hepatocyte growth factor receptor; Receptor tyrosine kinase that transduces signals from the extracellular matrix into the cytoplasm by binding to hepatocyte growth factor/HGF ligand. Regulates many physiological processes including proliferation, scattering, morphogenesis and survival. Ligand binding at the cell surface induces autophosphorylation of MET on its intracellular domain that provides docking sites for downstream signaling molecules. Following activation by ligand, interacts with the PI3-kinase subunit PIK3R1, PLCG1, SRC, GRB2, STAT3 or the adapter GAB1. Recruitment of thes [...]
    nextProt Receptor tyrosine kinase that transduces signals from the extracellular matrix into the cytoplasm by binding to hepatocyte growth factor/HGF ligand. Regulates many physiological processes including proliferation, scattering, morphogenesis and survival. Ligand binding at the cell surface induces autophosphorylation of MET on its intracellular domain that provides docking sites for downstream signaling molecules. Following activation by ligand, interacts with the PI3-kinase subunit PIK3R1, PLCG1, SRC, GRB2, STAT3 or the adapter GAB1. Recruitment of these downstream effectors by MET leads to the activation of several signaling cascades including the RAS-ERK, PI3 kinase-AKT, or PLCgamma-PKC. The RAS-ERK activation is associated with the morphogenetic effects while PI3K/AKT coordinates prosurvival effects. During embryonic development, MET signaling plays a role in gastrulation, development and migration of muscles and neuronal precursors, angiogenesis and kidney formation. In adults, participates in wound healing as well as organ regeneration and tissue remodeling. Promotes also differentiation and proliferation of hematopoietic cells. May regulate cortical bone osteogenesis (By similarity).
Interactions
NCBI
StringDB
Ontologies
Pathways
Transcripts
Accession Version MolecularType Name NCBI Comments
XM_011516223 2 mRNA transcript variant X1 NC_000007 Reference
NM_001127500 3 mRNA transcript variant 1 NC_000007 Reference
NM_000245 4 mRNA transcript variant 2 NC_000007 Reference
NM_001324402 2 mRNA transcript variant 4 NC_000007 Reference
NM_001324401 3 mRNA transcript variant 3 NC_000007 Reference
XM_047420400 1 mRNA transcript variant X2 NC_000007 Reference